| Imported post
A very interesting thread this......With some excellent, and very well informed posts. I'm certainly no expert on cardiorespiratory physiology but I have done some diving...... The primary "risk" associated with high PPO2 oxygen exposure is that of oxtox. The effect of CNS oxygen toxicity in diving is.....drowning. An O2 hit can result in a convultion which will *drown* you. That is the difference between hyperbaic medicine, where patients are regulrly exposed to PPO2'sof 2.8 bar or whatever, and diving, where we like to set a max ppO2 of 1.6 bar at deco and a much lower limit for the "working" portion of the dive. You can tox in a chamber quite "safely". Tox in the water and you need a really switched on buddy team to help you out. |